Предисловие

   1. Harach, H. R., K. O. Franssila, and V. M. Wasenius, “Occult Papillary Carcinoma of the Thyroid: A ‘Normal’ Finding in Finland: A Systematic Autopsy Study,” Cancer 56, no. 3 (1985): 531-38.

   2. Black, W. C., and H. G. Welch, “Advances in Diagnostic Imaging and Overestimations of Disease Prevalence and the Benefits of Therapy,” New England Journal of Medicine 328, no. 17 (1993): 1237-43.

   3. Stewart, B. W., and P. Kleihues, eds., World Cancer Report (Lyon, France: W.H.O. IARC Press, 2003).

   4. Yatani, R., T. Shiraishi, K. Nakakuki, et al., “Trends in Frequency of Latent Prostate Carcinoma in Japan from 1965-1979 to 1982-1986,” Journal of the National Cancer Institute 80, no. 9 (1988): 683-87.

   5. Stewart and Kleihues, World Cancer Report.

   6. Sorensen, T. I. A., G. G. Nielsen, P. K. Andersen, et al., “Genetic and Environmental Influences on Premature Death in Adult Adoptees,” New England Journal of Medicine 318 (1988): 727-32.

   7. Lichtenstein, P., N. V. Holm, P. K. Verkasalo, et al. “Environmental and Heritable Factors in the Causation of Cancer—Analyses of Cohorts of Twins from Sweden, Denmark, and Finland,” New England Journal of Medicine 343, no. 2 (2000): 78-85.

 

Предисловие ко второму изданию

   1. World Cancer Research Fund, Food, Nutrition and the Prevention of Cancer: A Global Perspective (London: World Cancer Research Fund and American Institute for Research on Cancer, 2007).

   2. Institut National du Cancer, Nutrition et prevéntion des cancers: des connaissances scientifiques aux recommandations (Paris: Ministere de la Santé et des Sports, 2009).

   3. Knoops, K. T. B., et al., “Mediterranean Diet, Lifestyle Factors, and 10-Year Mortality in Elderly European Men and Women—The HALE Project,” JAMA 292 (2004): 1433-39.

   4. Khaw, K.-T., et al., “Combined Impact of Health Behaviours and Mortality in Men and Women: The EPIC-Norfolk Prospective Population Study,” PLoS Medicine 5, no. 1 (2008), e12.

 

Глава 2

   1. Spiegel, D., “A 43-Year-Old Woman Coping with Cancer,” JAMA 282, no. 4 (1999): 371-78.

   2. Van Baalen, D. C., M. J. deVries, and M. T. Gondrie, “Psycho-social Correlates of ‘Spontaneous’ Regression in Cancer,” monograph, Department of General Pathology, Medical Faculty, Erasmus University, Rotterdam, The Netherlands, 1987.

   3. Lerner, M., oral communication, Smith Farm Retreat, 2001.

   4. Ornish, D., G. Weidner, W. R. Fair, et al., “Intensive Lifestyle Changes May Affect the Progression of Prostate Cancer,” Journal of Urology 174, no. 3 (2005): 1065-69, discussion 9-70.

   5. Ornish, D., M. J. Magbanua, G. Weidner, et al., “Changes in Prostate Gene Expression in Men Undergoing an Intensive Nutrition and Lifestyle Intervention,” Proceedings of the National Academy of Sciences 105 (on press): 8369-74.

   6. Ghadirian, P., S. Narod, E. Fafard, M. Costa, A. Robidoux, and A. Nkondjock, “Breast Cancer Risk in Relation to the Joint Effect of BRCA Mutations and Diet Diversity,” Breast Cancer Research & Treatment (2009).

   7. Fradet, V., I. Cheng, G. Casey, and J. S. Witte, “Dietary Omega-3 Fatty Acids, Cyclooxygenase-2 Genetic Variation, and Aggressive Prostate Cancer Risk,” Clinical Cancer Research 15 (2009): 2559-66.

   8. King, M.-C., J. H. Marks, J. B. Mandell, New York Breast Cancer Study Group, “Breast and Ovarian Cancer Risks Due to Inherited Mutations in BRCA1 and BRCA2,” Science 302 (2003): 643-46.

 

Глава 3

   1. Yalom, I., Existential Psychotherapy (New York: Basic Books, 1977). Есть перевод: Ирвин Ялом, Экзистенциальная психотерапия. Перевод Т.С.Драбкиной, М.: "Класс", 1999

   2. Ibid.

 

Глава 4

   1. Westcott, R., “Can Miracles Happen?” British Medical Journal 325, no. 7363 (2002): 553.

   2. Everson, T. C., “Spontaneous Regression of Cancer,” Progress in Clinical Cancer (1967): 79-95.

   3. Cole, W. H., “Efforts to Explain Spontaneous Regression of Cancer,” Journal of Surgical Oncology 17, no. 3 (1981): 201-9.

   4. Challis, G. B., H. J. Stam, G. B. Challis, et al., “The Spontaneous Regression of Cancer: A Review of Cases from 1900 to 1987,” Acta Oncologica 29, no. 5 (1990): 545-50.

   5. Bodey, B., B. Bodey, Jr., S. E. Siegel, et al., “The Spontaneous Regression of Neoplasms in Mammals: Possible Mechanisms and Their Application in Immunotherapy,” In Vivo 12, no. 1 (1998): 107-22.

   6. Papac, R. J., “Spontaneous Regression of Cancer: Possible Mechanisms,” In Vivo 12, no. 6 (1998): 571-78.

   7. Van Baalen, D. C., M. J. deVries, and M. T. Gondrie, “Psycho-social Correlates of ‘Spontaneous’ Regression in Cancer,” monograph, Department of General Pathology, Medical Faculty, Erasmus University, Rotterdam, The Netherlands, 1987.

   8. Cui, Z., M. C. Willingham, M. A. Alexander-Miller, et al., “Spontaneous Regression of Advanced Cancer: Identification of a Unique Genetically Determined, Age-Dependent Trait in Mice,” Proceedings of the National Academy of Sciences of the United States of America 100 (2003): 6682-87.

   9. Hicks, A. M., G. Riedlinger, M. C. Willingham, et al., “Transferable Anticancer Innate Immunity in Spontaneous Regression/Complete Resistance Mice,” Proceedings of the National Academy of Sciences of the United States of America 103, no. 20 (2006): 7753-58.

   10. Trapani, J. A., and M. J. Smyth, “Functional Significance of the Perforin/Granzyme Cell Death Pathway,” Nature Reviews Immunology 2 (2005): 735-47.

   11. Voskoboinik, I., and J. A. Trapani, “Addressing the Mysteries of Perforin Function,” Immunology and Cell Biology 84 (2006): 66-71.

   12. Whiteside, T., and R. B. Herberman, “Characteristics of Natural Killer Cells and Lymphocyte-Activated Killer Cells,” Immunology and Allergy Clinics of North America 10 (1990): 663-704.

   13. Head, J. F., F. Wang, R. L. Elliott, et al., “Assessment of Immunologic Competence and Host Reactivity Against Tumor Antigens in Breast Cancer Patients: Prognostic Value and Rationale of Immunotherapy Development,” Annals of the New York Academy of Sciences 690 (1993): 340-42.

   14. Levy, S. M., R. B. Herberman, M. Lippman, et al., “Immunological and Psychosocial Predictors of Disease Recurrence in Patients with Early-Stage Breast Cancer,” Behavioral Medicine 17, no. 2 (1991): 67-75.

   15. Imai, K., S. Matsuyama, S. Miyake, et al., “Natural Cytotoxic Activity of Peripheral-Blood Lymphocytes and Cancer Incidence: An 11-Year Follow-Up Study of a General Population,” Lancet 356, no. 9244 (2000): 1795-99.

   16. Schantz, S. P., B. W. Brown, E. Lira, et al., “Evidence for the Role of Natural Immunity in the Control of Metastatic Spread of Head and Neck Cancer,” Cancer Immunology, Immunotherapy 25, no. 2 (1987): 141-48.

   17. Herberman, R. B., “Immunotherapy,” in Clinical Oncology, ed. R. J. Lenhard, R. Osteen, and T. Gansler (Atlanta, GA: American Cancer Society, 2001), 215-23.

   18. MacKie, R. M., R. Reid, and B. Junor, “Fatal Melanoma Transferred in a Donated Kidney 16 Years after Melanoma Surgery,” New England Journal of Medicine 348, no. 6 (2003): 567-68.

   19. Cui, Z., “The Winding Road to the Discovery of the SR/CR Mice,” Cancer Immunity 3 (2003): 14.

   20. Koebel, C. M., Vermi, W., Swann, J. B., et al., “Adaptive Immunity Maintains Occult Cancer in an Equilibrium State,” Nature 159 (2008): 363-76.

   21. Imai, Matsuyama, Miyake, et al., “Natural Cytotoxic Activity of Peripheral-Blood Lymphocytes and Cancer Incidence.”

   22. Herberman, “Immunotherapy.”

   23. Levy, S. M., R. B. Herberman, A. M. Maluish, et al., “Prognostic Risk Assessment in Primary Breast Cancer by Behavioral and Immunological Parameters,” Health Psychology 4, no. 2 (1985): 99-113.

   24. Lutgendorf, S. K., A. K. Sood, B. Anderson, et al., “Social Support, Psychological Distress, and Natural Killer Cell Activity in Ovarian Cancer,” Journal of Clinical Oncology 23, no. 28 (2005): 7105-13.

   25. Schantz, Brown, Lira, et al., “Evidence for the Role of Natural Immunity in the Control of Metastatic Spread of Head and Neck Cancer.”

   26. Dvorak, H. F., “Tumors: Wounds That Do Not Heal: Similarities Between Tumor Stroma Generation and Wound Healing,” New England Journal of Medicine 315, no. 26 (1986): 1650-59.

   27. Balkwill, F., and A. Mantovani, “Inflammation and Cancer: Back to Virchow?” Lancet 357, no. 9255 (2001): 539-45.

   28. Peek, R. M., Jr., S. Mohla, and R. N. DuBois, “Inflammation in the Genesis and Perpetuation of Cancer: Summary and Recommendations from a National Cancer Institute-Sponsored Meeting,” Cancer Research 65, no. 19 (2005): 8583-86.

   29. Huang, M., M. Stolina, S. Sharma, et al., “Non-Small Cell Lung Cancer Cyclooxygenase-2-Dependent Regulation of Cytokine Balance in Lymphocytes and Macrophages: Up-Regulation of Interleukin 10 and Down-Regulation of Interleukin 12 Production,” Cancer Research 58, no. 6 (1998): 1208-16.

   30. Mantovani, A., B. Bottazzi, F. Colotta, et al., “The Origin and Function of Tumor-Associated Macrophages,” Immunology Today 13, no. 7 (1992): 265-70.

   31. Baxevanis, C. N., G. J. Reclos, A. D. Gritzapis, et al., “Elevated Prostaglandin E2 Production by Monocytes Is Responsible for the Depressed Levels of Natural Killer and Lymphokine-Activated Killer Cell Function in Patients with Breast Cancer,” Cancer 72, no. 2 (1993): 491-501.

   32. Marx, J., “Cancer Research: Inflammation and Cancer: The Link Grows Stronger,” Science 306 (2004): 5698-966.

   33. Wallace, J., “Nutritional and Botanical Modulation of the Inflammatory Cascade—Eicosanoids, Cyclooxygenases, and Lipoxygenases—as an Adjunct in Cancer Therapy,” Integrative Cancer Therapies 1, no. 1 (2002): 7-37.

   34. Crumley, A. B. C., D. C. McMillan, M. McKernan, et al., “Evaluation of an Inflammation-Based Prognostic Score in Patients with Inoperable Gastro-oesophageal Cancer,” British Journal of Cancer 94, no. 5 (2006): 637-41.

   35. Al Murri, A. M., J. M. S. Bartlett, P. A. Canney, et al., “Evaluation of an Inflammation-Based Prognostic Score (GPS) in Patients with Metastatic Breast Cancer,” British Journal of Cancer 94, no. 2 (2006): 227-30.

   36. Forrest, L. M., D. C. McMillan, C. S. McArdle, et al., “Comparison of an Inflammation-Based Prognostic Score (GPS) with Performance Status (ECOG) in Patients Receiving Platinum-Based Chemotherapy for Inoperable Non-Small-Cell Lung Cancer,” British Journal of Cancer 90, no. 9 (2004): 1704-6.

   37. Harris, R. E., S. Kasbari, and W. B. Farrar, “Prospective Study of Nonsteroidal Anti-inflammatory Drugs and Breast Cancer,” Oncology Reports 6, no. 1 (1999): 71-73.

   38. Nelson, J. E., and R. E. Harris, “Inverse Association of Prostate Cancer and Non-steroidal Anti-Inflammatory Drugs (NSAIDs): Results of a Case-Control Study,” Oncology Reports 7, no. 1 (2000): 169-70.

   39. Thun, M. J., “NSAID Use and Decreased Risk of Gastrointestinal Cancers,” Gastroenterology Clinics of North America 25, no. 2 (1996): 333-48.

   40. Karin, M., and F. R. Greten, “NF-kappaB: Linking Inflammation and Immunity to Cancer Development and Progression,” Nature Reviews Immunology 5, no. 10 (2005): 749-59.

   41. Marx, “Cancer Research.”

   42. Ibid.

   43. Calcagni, E., and I. Elenkov, “Stress System Activity, Innate and T Helper Cytokines, and Susceptibility to Immune-Related Diseases,” Annals of the New York Academy of Sciences 1069 (2006): 62-76.

   44. Glaser, R., “Stress-Associated Immune Dysregulation and Its Importance for Human Health: A Personal History of Psychoneuroimmunology,” Brain, Behavior, & Immunity 19, no. 1 (2005): 3-11.

   45. Beevor, A., Stalingrad: The Fateful Siege: 1942-1943 (New York: Penguin Group, 1998).

   46. Folkman, J., “Fighting Cancer by Attacking Its Blood Supply,” Scientific American , September 1996, 150-54.

   47. Folkman, J., “Tumor Angiogenesis: Therapeutic Implications,” New England Journal of Medicine 285, no. 21 (1971): 1182-86.

   48. Ibid.

   49.  “Cancer Warrior,” NOVA Online, 2001. (Accessed November 2, 2006, at

http://www.pbs.org/wgbh/nova/cancer/program.html.)

   50. O’Reilly, M. S., L. Holmgren, Y. Shing, et al., “Angiostatin: A Novel Angiogenesis Inhibitor That Mediates the Suppression of Metastases by a Lewis Lung Carcinoma,” Cell 79, no. 2 (1994): 315-28.

   51. O’Reilly, M. S., L. Holmgren, C. Chen, et al., “Angiostatin Induces and Sustains Dormancy of Human Primary Tumors in Mice,” Nature Medicine 2, no. 6 (1996): 689-92.

   52. Rose, D. P., and J. M. Connolly, “Regulation of Tumor Angiogenesis by Dietary Fatty Acids and Eicosanoids,” Nutrition and Cancer 37, no. 2 (2000): 119-27.

   53. Béliveau, R., and D. Gingras, Foods That Fight Cancer (New York: McClelland & Stewart Ltd., 2006).

   54. Béliveau, R., and D. Gingras, “Green Tea: Prevention and Treatment of Cancer by Nutraceuticals,” Lancet 364, no. 9439 (2004): 1021-22.

   55. Rose and Connolly, “Regulation of Tumor Angiogenesis by Dietary Fatty Acids and Eicosanoids.”

   56. Ziche, M., J. Jones, and P. M. Gullino, “Role of Prostaglandin E1 and Copper in Angiogenesis,” Journal of the National Cancer Institute 69, no. 2 (1982): 475-82.

 

Глава 6

   1. Dinse, G. E., D. M. Umbach, A. J. Sasco, et al., “Unexplained Increases in Cancer Incidence in the United States from 1975 to 1994: Possible Sentinel Health Indicators?” Annual Review of Public Health 20 (1999): 173-209.

   2. Institut de Veille Sanitaire, “Estimations Nationales: Tendances de l’Incidence et de la Mortalité par Cancer en France entre 1978 et 2000,” Ministère de la Santé, de la Famille et des Personnes Handicapées, 2002.

   3. Surveillance Epidemiology and End Results (SEER). Cancer incidence public use database, 2006; see http://seer.cancer.gov/.

   4. McGrath, K. G., “An Earlier Age of Breast Cancer Diagnosis Related to More Frequent Use of Antiperspirants/Deodorants and Underarm Shaving,” European Journal of Cancer Prevention 12, no. 6 (2003): 479-85.

   5. Steliarova-Foucher, E., C. Stiller, P. Kaatsch, et al., “Geographical Patterns and Time Trends of Cancer Incidence and Survival Among Children and Adolescents in Europe Since the 1970s (the ACCIS Project): An Epidemiological Study,” Lancet 364, no. 9451 (2004): 2097-2105.

   6. Post, P. N., D. Stockton, T. W. Davies, et al., “Striking Increase in Incidence of Prostate Cancer in Men Aged <60 Years Without Improvement in Prognosis,” British Journal of Cancer 79, no. 1 (1999): 13-17.

   7. Institut de Veille Sanitaire, “Estimations Nationales.”

   8. Ries, L. A. G., M. P. Eisner, C. L. Kosary, et al., “SEER Cancer Statistics Review 1975-2001,” National Cancer Institute, Bethesda, MD, 2004.

   9. Institut de Veille Sanitaire, “Estimations Nationales.”

   10. Ries, Eisner, Kosary, et al., “SEER Cancer Statistics Review 1975-2001.”

   11. Ferlay, J., F. Bray, P. Piesci, et al., eds., WHO International Agency for Research on Cancer (IARC), IARC Cancer Epidemiology Database, Globocan 2000, Cancer Incidence, Mortality and Prevalence Worldwide (Lyon, France: IARC Press, 2000).

   12. King, M-C., J. H. Marks, J. B. Mandell, et al., “Breast and Ovarian Cancer Risks Due to Inherited Mutations in BRCA1 and BRCA2,” Science 302, no. 5645 (2003): 643-46.

   13. Institut de Veille Sanitaire, “Estimations Nationales.”

   14. Rosenberg, C. E., The Cholera Years: The United States in 1832, 1849, and 1866 (Chicago, IL: University of Chicago Press, 1962).

   15. Steingraber, S., Living Downstream: A Scientist’s Personal Investigation of Cancer and the Environment (New York: Vintage Books, 1998).

   16. Davis, D., The Secret History of the War on Cancer (New York: Basic Books, 2007).

   17. Waterhouse, J., C. Muir, K. Shamnugaratnam, et al., eds., Cancer Incidence in Five Continents, vol. IV (Lyon, France: IARC-W.H.O., 1982).

   18. Sasco, A. J., “Migration and Cancer,” Revue de Médecine Interne 10, no. 4 (1989): 341-48.

   19. Davis, The Secret History of the War on Cancer.

   20. Waterhouse and Shamnugaratnam, Cancer Incidence in Five Continents.

   21. Stewart, B. W., and P. Kleihues, eds., World Cancer Report (Lyon, France: W.H.O. IARC Press, 2003).

   22. National Cancer Institute, Executive Summary of Cancer Etiology Think Tank (Bethesda, MD: National Cancer Institute, 2004).

   23. Eaton, S. B., and M. Konner, “Paleolithic Nutrition: A Consideration of Its Nature and Current Implications,” New England Journal of Medicine 312, no. 5 (1985): 283-89.

   24. Cordain, L., S. Eaton, A. Sebastian, et al., “Origins and Evolution of the Western Diet: Health Implications for the 21st Century,” American Journal of Clinical Nutrition 81, no. 2 (2005): 341-54.

   25. Ibid.

   26. Grothey, A., W. Voigt, C. Schober, et al., “The Role of Insulin-Like Growth Factor I and Its Receptor in Cell Growth, Transformation, Apoptosis, and Chemoresis tance in Solid Tumors,” Journal of Cancer Research & Clinical Oncology 125, no. 3-4 (1999): 166-73.

   27. Long, L., R. Navab, and P. Brodt, “Regulation of the Mr 72,000 Type IV Collagenase by the Type I Insulin-Like Growth Factor Receptor,” Cancer Research 58m, no. 15 (1998): 3243-47.

   28. Dunn, S. E., R. A. Hardman, F. W. Kari, et al., “Insulin-Like Growth Factor 1 (IGF-1) Alters Drug Sensitivity of HBL 100 Human Breast Cancer Cells by Inhibition of Apoptosis Induced by Diverse Anticancer Drugs,” Cancer Research 57, no. 13 (1997): 2687-93.

   29. Cordain, L., S. Lindeberg, M. Hurtado, et al., “Acne Vulgaris: A Disease of Western Civilization,” Archives of Dermatology 138, no. 12 (2002): 1584-90.

   30. Smith, R., N. Mann, A. Braue, et al., “The Effect of a Low Glycemic Load, High Protein Diet on Hormonal Markers of Acne,” Asia Pacific Journal of Clinical Nutrition 14 (supp.) (2005): S43.

   31. Smith, R., N. Mann, A. Braue, et al., “Low Glycemic Load, High Protein Diet Lessens Facial Acne Severity,” Asia Pacific Journal of Clinical Nutrition 14 (supp.) (2005): S97.

   32. Santisteban, G. A., J. T. Ely, E. E. Hamel, et al., “Glycemic Modulation of Tumor Tolerance in a Mouse Model of Breast Cancer,” Biochemical & Biopbysical Research Communications 132, no. 3 (1985): 1174-79.

   33. Parkin, D., F. Bay, J. Ferlay, et al., “Global Cancer Statistics, 2002,” CA: A Cancer Journal for Clinicians 55 (2005): 74-108.

   34. Weiderpass, E., G. Gridley, I. Persson, et al., “Risk of Endometrial and Breast Cancer in Patients with Diabetes Mellitus,” International Journal of Cancer 71, no. 3 (1997): 360-63.

   35. Hankinson, S. E., W. C. Willett, G. A. Colditz, et al., “Circulating Concentrations of Insulin-Like Growth Factor-I and Risk of Breast Cancer,” Lancet 351, no. 9113 (1998): 1393-96.

   36. Chan, J. M., M. J. Stampfer, E. Giovannucci, et al., “Plasma Insulin-Like Growth Factor-I and Prostate Cancer Risk: A Prospective Study,” Science 279, no. 5350 (1998): 563-66.

   37. Chan, J. M., M. J. Stampfer, J. Ma, et al., “Insulin-Like Growth Factor-I (IGF-I) and IGF Binding Protein-3 as Predictors of Advanced-Stage Prostate Cancer,” Journal of the National Cancer Institute 94, no. 14 (2002): 1099-1106.

   38. Michaud, D. S., S. Liu, E. Giovannucci, et al., “Dietary Sugar, Glycemic Load, and Pancreatic Cancer Risk in a Prospective Study,” Journal of the National Cancer Institute 94, no. 17 (2002): 1293-1300.

   39. Michaud, D. S., C. S. Fuchs, S. Liu, et al., “Dietary Glycemic Load, Carbohydrate, Sugar, and Colorectal Cancer Risk in Men and Women,” Cancer Epidemiology, Biomarkers & Prevention 14, no. 1 (2005): 138-47.

   40. Franceschi, S., L. Dal Maso, L. Augustin, et al., “Dietary Glycemic Load and Colorectal Cancer Risk,” Annals of Oncology 12, no. 2 (2001): 173-78.

   41. Augustin, L. S. A., J. Polesel, C. Bosetti, et al., “Dietary Glycemic Index, Glycemic Load and Ovarian Cancer Risk: A Case-Control Study in Italy,” Annals of Oncology 14, no. 1 (2003): 78-84.

   42. Gunter, M. J., et al., “Insulin, Insulin-Like Growth Factor-I, and Risk of Breast Cancer in Postmenopausal Women,” Journal of the National Cancer Institute 101 (2009): 48-60.

   43. McMillan-Price, J., et al., “Comparison of 4 Diets of Varying Glycemic Load on Weight Loss and Cardiovascular Risk Reduction in Overweight and Obese Young Adults: A Randomized Controlled Trial,” Archives of Internal Medicine 166, no. 14 (2006): 1466-75.

   44. Collectif LaNutrition.fr., Le Régime IG Minceur: comment perdre du poids en maîtrisant son sucre sanguin (Vergèze, France: Thierry Souccar Editions, 2007).

   45. Heini, A. F., and R. L. Weinsier, “Divergent Trends in Obesity and Fat Intake Patterns: The American Paradox,” American Journal of Medicine 102, no. 3 (1997): 259-64.

   46. Willett, W.C., “Dierary Fat Plays a Major Role in Obesity: No,” Obesity Reviews 3, no. 2 (2002): 59-68.

   47. Weill, P., Tous Gros Demain? (Paris, France: Plon, 2007).

   48. Ibid.

   49. Ailhaud, G., and P. Guesnet, “Fatty Acid Composition of Fats Is an Early Determinant of Childhood Obesity: A Short Review and an Opinion,” Obesity Reviews 5, no. 1 (2004): 21-26.

   50. Ailhaud, G., F. Massiera, P. Weill, et al., “Temporal Changes in Dietary Fats: Role of n-6 Polyunsaturated Fatty Acids in Excessive Adipose Tissue Development and Relationship to Obesity,” Progress in Lipid Research 45, no. 3 (2006): 203-36.

   51. Weill, P., B. Schmitt, G. Chesneau, et al., “Effects of Introducing Linseed in Livestock Diet on Blood Fatty Acid Composition of Consumers of Animal Products,” Annals of Nutrition & Metabolism 46, no. 5 (2002): 182-91.

   52. Ailhaud and Guesnet, “Fatty Acid Composition of Fats Is an Early Determinant of Childhood Obesity.”

   53. Simopoulos, A. P., “The Importance of the Ratio of Omega-6/Omega-3 Essential Fatty Acids,” Biomedicine Pharmacotherapy 56, no. 8 (2002): 365-79.

   54. Simopoulos, A. P., and N. Salem, “Omega-3 Fatty Acids in Eggs from Range-Fed Greek Chickens,” New England Journal of Medicine 321, no. 20 (1989): 1412.

   55. Ip, C., J. A. Scimeca, and H. J. Thompson, “Conjugated Linoleic Acid: A Powerful Anticarcinogen from Animal Fat Sources,” Cancer 74, 3 supp. (1994): 1050-54.

   56. Lavillonniere, F., V. Chajes, J-C. Martin, et al., “Dietary Purified cis-9, trans-11 Conjugated Linoleic Acid Isomer Has Anticarcinogenic Properties in Chemically Induced Mammary Tumors in Rats,” Nutrition and Cancer 45, no. 2 (2003): 190-94.

   57. Bougnoux, P., A. Barascu, M.-L. Jourdain, et al., “Acide Linoléique Conjugué et Cancer du Sein,” Oléagineux, Corps Gras, Lipides 2005; 12(1): 56-60.

   58. Dubnov, G., and E. M. Berry, “Omega-6/Omega-3 Fatty Acid Ratio: The Israeli Paradox,” World Review of Nutrition & Dietetics 92 (2003): 81-91.

   59. Weill, Tous Gros Demain?

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   61. Nationaal Kompas Volksgezondheid. “Verkeersongevallen. Omvang van het probleem. Verkeersongevallen naar leeftijd en geslacht, 2003-2007”: Public Health Department, Netherlands, 2004.

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   63. Hibbeln, J., W. Lands, and E. Lamoreaux, “Quantitative Changes in the Availability of Fats in the US Food Supply,” 5th Congress of the International Society for Study of Fatty Acids and Lipids, May 7-11, 2002 Montreal, Canada, 2002, p. 10.

   64. Bougnoux, P., et al., “ Alpha-Linolenic Acid Content of Adipose Breast Tissue: A Host Determinant of the Risk of Early Metastasis in Breast Cancer,” British Journal of Cancer 70, no. 2 (1994): 330-34.

   65. Maillard, V., P. Bougnoux, P. Ferrari, et al., “N-3 and N-6 Fatty Acids in Breast Adipose Tissue and Relative Risk of Breast Cancer in a Case-Control Study in Tours, France,” International Journal of Cancer 98, no. 1 (2002): 78-83.

   66. Pollan, M., “Power Steer,” New York Times Magazine, March 31, 2002.

   67. Pollan, M., “Unhappy Meals,” New York Times Magazine, January 28, 2007.

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   101. Kouba, M., “Quality of Organic Animal Products,” Lifestock Production Science 80 (2003): 33-40.

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   105. Ibid.

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Глава 7

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Глава 8

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Глава 10

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Глава 11

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   8. Meyerhardt, J. A., D. Heseltine, D. Niedzwiecki, et al., “Impact of Physical Activity on Cancer Recurrence and Survival in Patients with Stage III Colon Cancer: Findings from CALGB 89803,” Journal of Clinical Oncology 24, no. 22 (2006): 3535-41.

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   16. Irwin, M. L., “Randomized Controlled Trials of Physical Activity and Breast Cancer Prevention,” Exercise & Sport Sciences Reviews 34, no. 4 (2006): 182-93.

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   32. Meyerhardt, J. A., E. L. Giovannucci, M. D. Holmes, et al., “Physical Activity and Survival After Colorectal Cancer Diagnosis,” Journal of Clinical Oncology 24, no. 22 (2006): 3527-34.

   33. Meyerhardt, J. A., D. Heseltine, D. Niedzwiecki, et al., “Impact of Physical Activity on Cancer Recurrence and Survival in Patients with Stage III Colon Cancer: Findings from CALGB 89803,” Journal of Clinical Oncology 24, no. 22 (2006): 3535-41.

   34. Holmes, M. D., W. Y. Chen, D. Feskanich, et al., “Physical Activity and Survival After Breast Cancer Diagnosis,” JAMA 293, no. 20 (2005): 2479-86.

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   37. Patel, A. V., C. Rodriguez, E. J. Jacobs, et al., “Recreational Physical Activity and Risk of Prostate Cancer in a Large Cohort of U.S. Men,” Cancer Epidemiology, Biomarkers & Prevention 14, no. 1 (2005): 275-79.

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   41. Irwin, M. L., “Randomized Controlled Trials of Physical Activity and Breast Cancer Prevention,” Exercise & Sport Sciences Reviews 34, no. 4 (2006): 182-93.

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   43. Adams, S. A., C. E. Matthews, J. R. Hebert, et al., “Association of Physical Activity with Hormone Receptor Status: The Shanghai Breast Cancer Study,” Cancer Epidemiology, Biomarkers & Prevention 15, no. 6 (2006): 1170-78.

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Глава 12

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   7. Cunningham and Watson, “How Psychological Therapy May Prolong Survival . . .”

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   11. Walsh, R., Essential Spirituality: The Seven Central Practices to Awaken Heart and Mind (New York: John Wiley & Sons, 1999).

 

Глава 13

   1. Hambrecht, R., C. Walther, S. Mobius-Winkler, et al., “Percutaneous Coronary Angioplasty Compared with Exercise Training in Patients with Stable Coronary Artery Disease: A Randomized Trial,” Circulation 109, no. 11 (2004): 1371-78.

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   7. Ornish, D., G. Weidner, W. R. Fair, et al., “Intensive Lifestyle Changes May Affect the Progression of Prostate Cancer,” Journal of Urology 174, no. 3(2005): 1065-69; discussion 1069-70.

   8. Andersen, B. L., et al., “Psychologic Intervention Improves Survival for Breast Cancer Patients: A Randomized Clinical Trial,” Cancer 113 (2008): 3450-58.

   9. Ballard-Barbash, R. and A. McTiernan, “Is the Whole Larger Than the Sum of the Parts? The Promise of Combining Physical Activity and Diet to Improve Cancer Outcomes,” Journal of Clinical Oncology 25, no. 17 (2007): 2335-2337.

   10. Pierce, J. P., et al., “Greater Survival After Breast Cancer in Physically Active Women with High Vegetable-Fruit Intake Regardless of Obesity,” Journal of Clinical Oncology 25, no. 17 (2007): 2345-51.

   11. World Cancer Research Fund, Food, Nutrition and the Prevention of Cancer: A Global Perspective (London: World Cancer Research Fund and American Institute for Research on Cancer, 2007), xxiii.